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NurseSavvy Cheat SheetDisease

Emphysema

Emphysema is permanent, irreversible destruction of the alveolar walls distal to the terminal bronchioles. Loss of elastic recoil means the alveoli cannot spring back on expiration, so air becomes trapped, the lungs hyperinflate, and gas-exchange surface area shrinks. Unlike chronic bronchitis (an airway/mucus disease), emphysema destroys lung architecture itself.

Pink puffer (emphysema) vs blue bloater (chronic bronchitis)

Emphysema (pink puffer)Chronic bronchitis (blue bloater)
Body habitusThin, cachecticOverweight, edematous
Cough / sputumMinimal, scantChronic productive cough, copious sputum
CyanosisMinimal earlyMarked (central cyanosis)
Breath soundsDiminishedCoarse crackles, rhonchi
Chest shapeBarrel chestOften normal
Primary problemAlveolar destructionAirway inflammation + mucus

Emphysema (pink puffer)

Body habitus
Thin, cachectic
Cough / sputum
Minimal, scant
Cyanosis
Minimal early
Breath sounds
Diminished
Chest shape
Barrel chest
Primary problem
Alveolar destruction

Chronic bronchitis (blue bloater)

Body habitus
Overweight, edematous
Cough / sputum
Chronic productive cough, copious sputum
Cyanosis
Marked (central cyanosis)
Breath sounds
Coarse crackles, rhonchi
Chest shape
Often normal
Primary problem
Airway inflammation + mucus

Diagnostic

decreased FEV1/FVC ratio
< 70% confirms obstruction
increased residual volume Hallmark
air trapping; goes UP, not down
increased total lung capacity
reduced DLCO
loss of alveolar surface area
hyperinflation on chest x-ray
flattened diaphragm, increased AP diameter

Monitor

preserved PaCO2 early
pink puffers hyperventilate for years
progressive hypoxemia
monitor oxygenation trends
assess work of breathing
low-flow supplemental oxygen
titrate to target SpO2; avoid oversedating hypoxic drive
position upright / forward-leaning
pace activity to tolerance
high-calorie nutrition support
counter cachexia from breathing effort
inhaled bronchodilators
inhaled corticosteroids
smoking cessation pharmacotherapy
nicotine replacement, varenicline
pulmonary rehabilitation
smoking cessation at any stage
quitting slows FEV1 decline even late
pursed-lip breathing technique
high-calorie small frequent meals
energy conservation strategies
pneumococcal and influenza vaccination
Report Nowescalate immediately
paradoxical breathing
diaphragmatic fatigue; impending respiratory failure
rising PaCO2 with pH < 7.30pH < 7.30
acute ventilatory failure on chronic CO2 retention
declining mental status
CO2 narcosis or severe hypoxemia
acute worsening dyspnea
acute exacerbation

Clinical Pearl

Emphysema lungs are an old stretched-out balloon: air goes in easily, but the walls lost their snap — so air stays trapped and the client struggles to breathe OUT, not in.

NurseSavvy™·nursesavvy.com

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