multi class comparison

RAAS Drugs: ACEi vs ARBs vs Direct Renin Inhibitor

A client on lisinopril develops a persistent dry cough — do you hold the drug, switch classes, or reassure? Picking the wrong answer means you don't know which side effect belongs to which drug class. The NCLEX exploits this confusion repeatedly across pharmacology and safety questions.

Comparison

Side-by-side3 compared
Comparevs
Dimension
ACE Inhibitors
ARBs
Direct Renin Inhibitor
Class & mechanism
  • Block ACE → ↓ angiotensin II, ↑ bradykinin
  • Block AT1 receptor → ↓ angiotensin II effect
  • Inhibit renin directly → ↓ angiotensin I
Indications
  • Hypertension
  • Heart failure; diabetic nephropathy
  • Hypertension
  • Heart failure; diabetic nephropathy
  • Hypertension
Route & suffix
  • PO; –pril (lisinopril, enalapril)
  • PO; –sartan (losartan, valsartan)
  • PO; aliskiren
Key assessment
  • Blood pressure
  • Dry cough (bradykinin)
  • Blood pressure
  • No cough — alternative when ACEi cough
  • Blood pressure
  • Diarrhea (high doses)
Monitoring labs
  • Monitor potassium & renal function
  • Monitor potassium & renal function
  • Monitor potassium & renal function
Adverse effects
  • Hyperkalemia — avoid K⁺ supplements/salt substitutes
  • Dry cough + angioedema (bradykinin)
  • Hyperkalemia — avoid K⁺ supplements/salt substitutes
  • Angioedema rare
  • Hyperkalemia — avoid K⁺ supplements/salt substitutes
  • Diarrhea; angioedema
Black box & emergency
  • Pregnancy — fetal toxicity (black box)
  • Angioedema — airway emergency, stop drug
  • Pregnancy — fetal toxicity (black box)
  • Angioedema — airway emergency, stop drug
  • Pregnancy — fetal toxicity (black box)
  • Angioedema — airway emergency, stop drug
Contraindications & interactions
  • Avoid with K-sparing diuretics
  • Avoid with K-sparing diuretics
  • Avoid with K-sparing diuretics
Patient teaching
  • Rise slowly (first-dose hypotension)
  • Rise slowly (first-dose hypotension)
  • Rise slowly (first-dose hypotension)
Class & mechanism

ACE Inhibitors

  • Block ACE → ↓ angiotensin II, ↑ bradykinin

ARBs

  • Block AT1 receptor → ↓ angiotensin II effect
Indications

ACE Inhibitors

  • Hypertension
  • Heart failure; diabetic nephropathy

ARBs

  • Hypertension
  • Heart failure; diabetic nephropathy
Route & suffix

ACE Inhibitors

  • PO; –pril (lisinopril, enalapril)

ARBs

  • PO; –sartan (losartan, valsartan)
Key assessment

ACE Inhibitors

  • Blood pressure
  • Dry cough (bradykinin)

ARBs

  • Blood pressure
  • No cough — alternative when ACEi cough
Monitoring labs

ACE Inhibitors

  • Monitor potassium & renal function

ARBs

  • Monitor potassium & renal function
Adverse effects

ACE Inhibitors

  • Hyperkalemia — avoid K⁺ supplements/salt substitutes
  • Dry cough + angioedema (bradykinin)

ARBs

  • Hyperkalemia — avoid K⁺ supplements/salt substitutes
  • Angioedema rare
Black box & emergency

ACE Inhibitors

  • Pregnancy — fetal toxicity (black box)
  • Angioedema — airway emergency, stop drug

ARBs

  • Pregnancy — fetal toxicity (black box)
  • Angioedema — airway emergency, stop drug
Contraindications & interactions

ACE Inhibitors

  • Avoid with K-sparing diuretics

ARBs

  • Avoid with K-sparing diuretics
Patient teaching

ACE Inhibitors

  • Rise slowly (first-dose hypotension)

ARBs

  • Rise slowly (first-dose hypotension)

marks the fact that sets a column apart.

Clinical Pearl

Cough → ACEi; switch to ARB (no cough, no bradykinin). Aliskiren (DRI) blocks renin at the top of the cascade — never combine it with an ACEi or ARB in diabetes (ALTITUDE). Suffix tells the class: -pril, -sartan, aliskiren.

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