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NurseSavvy Cheat SheetDisease

Spinal Shock & Neurogenic Shock

Two distinct phenomena that can coexist after acute spinal cord injury. Spinal shock is the temporary loss of all reflex activity below the injury — flaccid paralysis and areflexia — a neurological state, not a hemodynamic emergency, that resolves over days to weeks. Neurogenic shock is a distributive shock from loss of sympathetic tone (injuries at T6 or above), so blood pools peripherally with no compensatory vasoconstriction.

Spinal shock vs Neurogenic shock

Spinal shockNeurogenic shock
NatureTransient loss of all reflexes (neurological)Distributive shock from lost sympathetic tone
Hallmark findingsFlaccid paralysis + areflexia below injuryHypotension + bradycardia + warm, dry, flushed skin
Hemodynamic emergency?No — not a shock stateYes — true shock requiring resuscitation
SettingAny level of cord injuryInjury at T6 or above
CourseResolves over days to weeksTreated until vascular tone supported
Resolution markerReturn of bulbocavernosus reflexRestored MAP and perfusion

Spinal shock

Nature
Transient loss of all reflexes (neurological)
Hallmark findings
Flaccid paralysis + areflexia below injury
Hemodynamic emergency?
No — not a shock state
Setting
Any level of cord injury
Course
Resolves over days to weeks
Resolution marker
Return of bulbocavernosus reflex

Neurogenic shock

Nature
Distributive shock from lost sympathetic tone
Hallmark findings
Hypotension + bradycardia + warm, dry, flushed skin
Hemodynamic emergency?
Yes — true shock requiring resuscitation
Setting
Injury at T6 or above
Course
Treated until vascular tone supported
Resolution marker
Restored MAP and perfusion
Hypotension Hallmark
neurogenic shock triad
Bradycardia Hallmark
distinguishes from hypovolemic shock
Warm, dry, flushed skin Hallmark
below injury; opposite of cold/clammy hypovolemia
Flaccid paralysis below injury
spinal shock
Absent deep tendon reflexes
spinal shock areflexia
Loss of bladder and bowel tone
spinal shock
Continuous blood pressure monitoring
Continuous cardiac monitoring
detect bradycardia
Bulbocavernosus reflex assessment
return signals spinal shock resolving
Serial neurological exam
motor/reflex tracking below injury
IV fluid resuscitation first
cautious; volume before pressors
Vasopressors if fluids fail
restore vascular tone
Atropine for symptomatic bradycardia
Maintain MAP 85-90 mmHgMAP 85-90 mmHg x 7 days
preserve cord perfusion first 7 days
Rule out concurrent hemorrhage
tachycardia suggests hidden hypovolemia
Norepinephrine
alpha-1 vasoconstriction; first-line per source question
Phenylephrine
alternative vasopressor
Atropine
for symptomatic bradycardia
Isotonic IV fluids
normal saline volume resuscitation
Autonomic dysreflexia
late complication after spinal shock resolves
Permanent neurological deficit
assessed only after spinal shock resolves
Report Nowescalate immediately
Persistent hypotension after fluids
raises suspicion for neurogenic cause or bleeding
Symptomatic bradycardia
needs atropine
Respiratory compromise in high SCI
cervical injury threatens diaphragm
Tachycardia with hypotension
hypovolemic hemorrhage hiding underneath

Clinical Pearl

Bradycardia + hypotension + warm skin after spinal cord injury = neurogenic shock. If the patient is tachycardic, keep looking for bleeding — that's hypovolemic shock hiding underneath.

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