Increased ICP — Monitoring & Interventions

Once increased ICP is identified, nursing interventions aim to reduce intracranial volume across three compartments: brain tissue, blood, and CSF. Position the head of bed at 30 degrees with the head in midline — turning the neck kinks the jugular veins and blocks venous outflow, spiking ICP further. Maintain a quiet, dim environment and cluster care to avoid repeated stimulation. Avoid hip flexion, Valsalva maneuvers (straining, coughing), and suctioning beyond 10 seconds per pass — each triggers transient ICP spikes. Maintain normothermia — fever increases cerebral metabolic demand and worsens ICP; use antipyretics and cooling measures as needed. Hyperosmotic agents are the pharmacologic frontline: mannitol (0.25–1 g/kg IV) draws fluid from brain tissue into the vascular space. Monitor serum osmolality — hold mannitol if osmolality exceeds 320 mOsm/kg to prevent renal failure. Hypertonic saline (3% NaCl) is an alternative that expands plasma volume without the osmotic rebound risk. Monitor sodium closely; target varies by protocol but generally stays below 160 mEq/L. An external ventricular drain (EVD) both monitors ICP numerically (normal 5–15 mmHg) and therapeutically drains CSF. Keep the drainage system leveled at the tragus of the ear (the external landmark approximating the foramen of Monro). Before repositioning the patient: clamp the drain → reposition → relevel at the tragus → unclamp. Maintain cerebral perfusion pressure (CPP = MAP − ICP) above 60 mmHg — below this threshold, ischemia begins.