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NurseSavvy Cheat SheetDisease

Burn Complications

Major burns (>20% TBSA) trigger a hypermetabolic stress response and massive capillary leak whose complications follow a predictable clock: airway edema first, eschar constriction within hours, Curling's ulcer in days, sepsis in weeks, contractures in months.

Burn complication clock

  1. Hours: airway edemainhalation injury -> intubate early
  2. Hours: eschar constrictionescharotomy for circulation/ventilation
  3. Days: Curling's ulcerstress-ulcer GI bleed
  4. Weeks: sepsisleading cause of death after resuscitation
  5. Months: contracturesscar shortening across joints
EarlyProgresses →
Singed nasal hairs Hallmark
objective inhalation-injury sign
Carbonaceous sputum Hallmark
soot in mouth/oropharynx
Hoarse voice
supraglottic edema marker
Facial burns
Late / Severe
Stridor
near-complete obstruction - act before this
Other findings
Cherry-red skin
carbon monoxide poisoning
Weakening distal pulses
circumferential-burn eschar compromise
Finger numbness and tingling
ischemia from constricting eschar
Coffee-ground NG drainage
Curling's ulcer bleed
Tea-colored urine Hallmark
myoglobinuria from rhabdomyolysis
Green foul-smelling wound drainage
Pseudomonas wound infection

Diagnostic

Co-oximetry Hallmark
measures carboxyhemoglobin; pulse ox cannot

Monitor

Carboxyhemoglobin levelCOHb < 5%
goal before weaning 100% FiO2
Pulse oximetry caveat
falsely normal/high in CO poisoning
Elevated hematocrit
hemoconcentration = burn shock, first 24-48h
Serum glucose
new hyperglycemia signals sepsis
WBC trend
falling WBC + hypothermia = sepsis
Gastric pH
low pH with bleed = inadequate prophylaxis
Hourly urine output
perfusion and resuscitation marker
Secure airway early Hallmark
intubate before edema closes airway
Apply 100% oxygen non-rebreather
maintain until COHb < 5%
Fluid resuscitation
counters capillary leak / burn shock
Assist bedside escharotomy
restores distal circulation and chest excursion
Increase IV fluids for myoglobinuriaurine output 75-100 mL/hr
flush myoglobin; do NOT restrict fluids
Infection-control precautions
sepsis is leading post-resuscitation killer
Early splinting
contracture prevention
Range-of-motion exercises
preserve joint mobility
Pantoprazole
PPI stress-ulcer prophylaxis for Curling's
Proton pump inhibitor
suppresses gastric acid
H2 receptor blocker
alternative acid suppression
Early enteral feeding Hallmark
within 6-12h; preserves gut mucosa, reduces translocation
Report worsening hoarseness or breathing
airway can deteriorate after admission
Adhere to splint and ROM schedule
prevents disabling contractures
Watch wounds for infection signs
increasing pain, odor, green drainage, fever
Maintain high-protein high-calorie intake
supports hypermetabolic healing
Burn shock
hypovolemia from capillary leak, first 48h
Curling's ulcer
burn stress ulcer; NOT Cushing's (neuro)
Burn wound sepsis
leading cause of death post-resuscitation
Acute kidney injury
hypoperfusion and myoglobinuria
Joint contractures
late, during rehabilitation
Report Nowescalate immediately
Impending airway obstruction Hallmark
hoarseness + singed hairs + soot; intubate before stridor
Carbon monoxide poisoning
cherry-red skin, confusion, falsely normal SpO2
Absent or weakening distal pulse
circumferential eschar - escharotomy
Rising peak airway pressures
circumferential chest eschar - chest escharotomy
Falling urine output< 0.5 mL/kg/hr
hypoperfusion / AKI
Early sepsis signs
hypothermia, leukopenia, new confusion, hyperglycemia
Coffee-ground NG drainage
Curling's ulcer GI bleed

Clinical Pearl

Airway in hours, eschar in hours, Curling's in days, sepsis in weeks, contractures in months - the burn complication clock never stops ticking.

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