Esophageal Varices
Pathophysiology & Risk Factors
Portal hypertension from liver cirrhosis forces blood into collateral venous pathways, including the thin-walled submucosal veins of the esophagus. These vessels were never built for high-pressure flow, so they dilate and become extremely fragile. Rupture produces sudden, massive, PAINLESS hematemesis that can be fatal within minutes.
Signs & Symptoms
Diagnostics & Labs
Diagnostic
Monitor
Interventions & Priorities
Active variceal bleed priorities
- Airwayprotect first; aspiration risk
- Accesstwo large-bore IVs
- Volume + bloodrestrictive resuscitation
- Octreotidelowers portal pressure
- Antibioticsprophylaxis
- Endoscopic band ligationdefinitive
Treatments & Medications
Patient Teaching
Complications
Clinical Pearl
Blood in the gut is protein in the gut. After a variceal bleed, think lactulose -- the cirrhotic liver can't clear the ammonia, and hepatic encephalopathy is the next crisis waiting to happen.